News reports of research into Alzheimer's disease July - December 2006

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There's a glossary of terms used in Alzheimer's research.

Disclaimer:
This section began as an offshoot of my gathering of news items about memory. I am not a medical expert. My background is in psychology. The information I have gathered here should not be taken as providing any advice.

December

New way to target Alzheimer's disease

In a series of studies in transgenic mice, a synthetic peptide designed to block the interaction between apolipoprotein E and amyloid-beta protein reduced the aggregation of toxic amyloid protein in the brain by around 50%. The treated mice showed no memory decline.
The study was published in the December 5 issue of the Proceedings of the National Academy of Sciences. Full reference
The full text is available at http://www.pnas.org/cgi/content/full/103/49/18787 http://www.eurekalert.org/pub_releases/2006-12/nyum-sda120406.htm

Androgen therapy may slow progress of Alzheimer's disease

Recent studies have suggested a link between testosterone loss in men and Alzheimer’s. A new study has now found a correlation between low testosterone and elevated beta-amyloid, providing more support that testosterone depletion in aging men increases the risk of Alzheimer’s. Testosterone belongs to a group of steroid hormones called androgens. The mouse study found that androgen therapy was successful in preventing beta-amyloid accumulation and cognitive decline in castrated mice.
The findings were published in the December 20 issue of The Journal of Neuroscience. Full reference
http://www.eurekalert.org/pub_releases/2006-12/sfn-tm121906.htm

Compound shows promise for early detection of Alzheimer's disease

A new molecular marker called FDDNP has been found to track the progression of Alzheimer’s in PET scans more effectively than other markers, giving hope of earlier, more accurate diagnosis of the disease.
The study was published in the December 21 issue of the New England Journal of Medicine. Full reference
http://www.eurekalert.org/pub_releases/2006-12/nioa-nic121906.htm
http://www.eurekalert.org/pub_releases/2006-12/uoc--nit121506.htm

Protein 'fingerprint' in spinal fluid could spot Alzheimer's disease early

In a pilot study, a panel of 23 protein biomarkers in cerebrospinal fluid has been found to be over 90% sensitive in identifying people with Alzheimer's disease.
The research was published online December 13 in Annals of Neurology. Full reference
http://www.eurekalert.org/pub_releases/2006-12/cuns-pi120706.htm

Hypertension drugs might help Alzheimer’s

A project to determine whether drugs that are already commercially available for treatment of other disorders might help in treating Alzheimer’s disease using in vitro methods has identified several hundred drugs as having promise in preventing beta-amyloid build-up, of which seven are commonly prescribed to treat hypertension. One drug in particular was identified as effective in blocking the accumulation of beta-amyloid in the brain and preventing the deterioration of cognitive performance: Propranololo-HCL (Inderal), a drug widely prescribed to treat high blood pressure in elderly patients. Of course, clinical trials will need to occur before this can be confirmed.
The research was reported at the American College of Neuropsychopharmacology’s (ACNP) annual conference December 3 - 7, 2006, in Hollywood, FL.
http://www.eurekalert.org/pub_releases/2006-12/g-nsf120106.htm

November

Higher level of certain fatty acid associated with lower dementia risk

A nine year study of 899 participants in the Framingham Heart Study (average age 76 years) has found that those with the highest levels of an omega-3 polyunsaturated fatty acid known as docosahexaenoic acid (DHA) had a 47% lower risk of developing dementia and 39% lower risk of developing Alzheimer's. Among the participants who completed the dietary questionnaire, those in this top quartile of blood DHA levels reported that they ate an average of .18 grams of DHA a day and an average of three fish servings a week. Those in the other quartiles ate substantially less fish.
The report appeared in the November issue of Archives of Neurology. Full reference
http://www.eurekalert.org/pub_releases/2006-11/jaaj-hlo110906.htm

Memory complaints early warning for Alzheimer's

A post-mortem study of 90 older adults from the Rush Memory and Aging Project found that those who had yet to have any clinical symptoms of Alzheimer's disease still showed a strong link between their self-reported memory complaints and brain pathology associated with Alzheimer's disease.
The findings were reported in the November issue of Neurology. Full reference
http://www.eurekalert.org/pub_releases/2006-11/rumc-cam113006.htm

Counseling Alzheimer's caregivers postpones the nursing home

A long-running study of 406 people caring for a spouse with dementia has found that a program of individual and family counseling sessions and ongoing support for caregivers delays by an average of 1.5 years the time when people with dementia are usually put in nursing homes.
The study was published in the November 14 issue of Neurology. Full reference
http://www.eurekalert.org/pub_releases/2006-11/nyum-cac110806.htm

Individualized counseling helps caregivers

A study of 642 people who were caring for a relative with Alzheimer's disease or a related disorder has found that those who participated in a counseling program were less likely to suffer depression and felt more confident and able to deal with caring for their relative. Hispanic and white participants saw the greatest benefit. In African-Americans, the intervention was effective among spouse-caregivers, but relatively ineffective among caregivers who were caring for a relative other than their husband or wife. Hispanics had the greatest improvement in reduction of caregiver depressive symptoms and problem behaviors of the recipient. Whites saw the most impact in the area of social support and African-American spouse-caregivers had the most positive outcome in reducing the caregiver burden and improving self-care.
Results were published in the November 21 issue of Annals of Internal Medicine. Full reference
http://www.eurekalert.org/pub_releases/2006-11/uopm-idc111506.htm
http://www.eurekalert.org/pub_releases/2006-11/nioa-npe111606.htm

New dementia screening tool detects early cognitive problems

A new screening tool for dementia — the Saint Louis University Mental Status Examination (SLUMS) — appears to work better in identifying mild cognitive problems in the elderly than the commonly used Mini Mental Status Examination — particularly for the more educated patients. It takes a clinician about seven minutes to administer the SLUMS, which supplements the Mini Mental Status Examination by asking patients to perform tasks such as doing simple math computations, naming animals, recalling facts and drawing the hands on a clock. The SLUMS is available at this link http://medschool.slu.edu/agingsuccessfully/pdfsurveys/slumsexam_05.pdf
The study appeared in the November issue of the American Journal of Geriatric Psychiatry. Full reference
http://www.eurekalert.org/pub_releases/2006-11/slu-nds103006.htm

What nerve fibers need to form connections

A discovery that cholinergic neuronal circuits rely on neurotrophin-3, a chemical that stimulates nerve growth, suggests a new approach to a therapy for Alzheimer’s. The brains of Alzheimer's patients show a marked decline in these cholinergic circuits, suggesting that a neurotrophin-3 therapy may be beneficial.
Study results appeared in the December 1 issue of Neuroscience. Full reference
http://www.eurekalert.org/pub_releases/2006-11/uoc--nfn111706.htm

October

Marijuana may slow progression of Alzheimer's disease

New evidence suggests that marijuana — which has strong anti-inflammatory effects — may contain compounds that slow the memory loss associated with Alzheimer's disease. Treatment with a synthetic compound similar to marijuana (WIN-55212-2) reduced inflammation in older rats and substantially improved their memories.
The researchers presented their findings October 18 at the annual Society for Neuroscience meeting in Atlanta.
http://www.eurekalert.org/pub_releases/2006-10/osu-lb101206.htm
http://www.sciencedaily.com/releases/2006/10/061018151055.htm

Anesthetics a risk factor for Alzheimer’s?

The link between surgery and cognitive problems has long been noted, but it’s never been clear whether postoperative cognitive dysfunction was the result of the surgery itself or the anaesthetics. Now animal studies and test tube experiments are beginning to show that certain anaesthetics reduce the rate at which brain cells are born and develop. The latest study reveals that the inhaled anaesthetics halothane and isoflurane encourage clumping of beta amyloid protein, as does the commonly used intravenous anaesthetic propofol, at least at higher concentrations — suggesting that giving elderly patients certain general anaesthetics could increase their risk of developing Alzheimer's disease. The intravenous anaesthetic thiopental appears to have no effect on the proteins.
The study was presented at the annual Society for Neuroscience Meeting held in Atlanta, Georgia, October 14-18.
http://www.newscientist.com/channel/health/mg19225753.900-alzheimers-alert-over-anaesthetics.html
http://www.eurekalert.org/pub_releases/2006-10/ns-aao102506.htm

Omega-3 fatty acids may slow cognitive decline in some patients with very mild Alzheimer's disease

Several studies have shown that eating fish, which is high in omega-3 fatty acids, may protect against Alzheimer's disease. A Swedish study has now tested whether supplements could have similar effects. Patients with mild-to-moderate Alzheimer’s who took 1.7 grams of DHA and .6g of EPA showed the same rate of cognitive decline as those taking a placebo, however, among a subgroup of 32 patients with very mild cognitive impairment, those who took the fatty acids experienced less decline in six months compared with those who took placebo. It may be that anti-inflammatory effects are an important reason for the benefit, potentially explaining why effects were seen only in those with very early-stage disease, when levels of inflammation seem to be higher.
The study was published in the October issue of Archives of Neurology. Full reference
http://www.eurekalert.org/pub_releases/2006-10/jaaj-ofa100506.htm

Computer-based 'games' enhance mental function in Alzheimer's patients

An interactive multimedia internet-based game has been shown to benefit cognition in patients with Alzheimer's disease more than classic exercises of mental stimulation commonly used with dementia patients. The study compared patients receiving no cognitive intervention, those enrolled in a daily program that included 2.5 to 3.5 hours of cognitive stimulation tasks, musical therapy, arts and crafts, physical activity and programs that reinforced instrumental activities of daily living, and those who also used an interactive multimedia internet-based system which allowed them to carry out a variety of different cognitive stimulation tasks at varying levels of difficulty throughout the day. After 12 weeks, both intervention groups performed better on tests than the control group; at 24 weeks, the dual-intervention group did better than the program-only group. The study points to the value of cognitive stimulation to slow the rate of cognitive loss.
The findings are published in the October issue of the Journal of Neurology, Neurosurgery and Psychiatry. Full reference
http://www.eurekalert.org/pub_releases/2006-10/uopm-ce102306.htm

Cabernet sauvignon red wine reduces the risk of Alzheimer's disease

A mouse study has found moderate consumption of the red wine Cabernet Sauvignon significantly reduced Alzheimer’s-type deterioration of spatial memory function. The Cabernet Sauvignon used contained a very low content of resveratrol, 10-fold lower than the minimal effective concentration shown to promote Aß clearance in vitro. It is suggested that, instead, the benefit occurred through promoting non-amyloidogenic processing of amyloid precursor protein. The finding supports epidemiological evidence indicating that moderate wine consumption (one drink per day for women and two for men) may help reduce the relative risk for Alzheimer’s.
The study was presented at the annual Society for Neuroscience Meeting held in Atlanta, Georgia, October 14-18, and will be published in the November issue of The FASEB Journal. Full reference
http://www.eurekalert.org/pub_releases/2006-09/tmsh-csr091806.htm

September

New early diagnostic test trialed

A mouse study has used a laser scan of the eyes to accurately diagnose Alzheimer's well before the disease was evident in the brain. The study follows on from earlier research revealing that beta-amyloid protein is evident in the eyes of Alzheimer’s patients. The test, which is a very quick and simple procedure, is now in the first stage of experimental trials in people.
The findings were announced at the annual meeting of the Optical Society of America.
http://www.sciencentral.com/articles/view.htm3?article_id=218392859

Insulin receptor stops progression of Alzheimer's

Following previous research suggesting Alzheimer's might be a brain-specific neuroendocrine disorder, or a Type 3 diabetes, a new study has found that stimulation of a receptor in the brain that controls insulin responses prevents several components of neurodegeneration and preserves learning and memory in rats with induced Alzheimer's disease, raising the possibility that patients in the very early stages of Alzheimer’s might be treatable.
The study appeared in the September issue of the Journal of Alzheimer's Disease. Full reference
http://www.eurekalert.org/pub_releases/2006-09/l-irs092106.htm

Link between increased weight-loss rate and dementia

Confirming earlier indications, a long-term study of the elderly has revealed that their average rate of weight loss doubles (from 0.6 pounds per year to 1.2 pounds per year) in the year before symptoms of Alzheimer's-type dementia first become detectable. The finding may be useful as one of several early biomarkers. The study analyzed data on 449 seniors, of whom 125 were eventually diagnosed with mild dementia. Interestingly, at the beginning of the study, this group weighed about 8lb less on average than the other participants, although the two groups lost weight at the same rate for four to five years, before weight loss increased in the group that would eventually be diagnosed with mild dementia. It is not yet known why there should be this connection between weight loss and dementia.
The report appeared in the September issue of Archives of Neurology. Full reference
http://www.eurekalert.org/pub_releases/2006-09/wuso-bdf090806.htm

Two-fold role of Alzheimer’s genes?

The genes responsible for an inherited form of Alzheimer's disease — two genes known as presenilins — are primarily known for their role as an enzyme that cleaves amyloid precursor protein (APP) to form amyloid ß-peptide, which function has a direct connection to Alzheimer’s, and consequently has been the focus of attention. However, new research indicates that these genes also may control the balance of calcium within cells by acting as a calcium channel, and that the mutated forms of these genes lose this ability. Given the role that calcium signaling plays in cognitive function, it may be that this other role of presenilins is also important in the development of Alzheimer’s. If so, drugs that restore normal calcium levels might be useful for treating Alzheimer's disease.
The report appeared in the September 8 issue of Cell. Full reference
http://www.eurekalert.org/pub_releases/2006-09/cp-ssa090106.htm

August

Juices may reduce Alzheimer's disease risk

In a large epidemiological study, that followed 1836 Seattle residents for up to 10 years, it was found that those who drank three or more servings of fruit and vegetable juices per week had a 76% lower risk of developing Alzheimer’s disease than those who drank juice less than once a week. The benefit seemed greatest for those who carried the so-called “Alzheimer’s gene”. Previously, researchers suspected that antioxidant vitamins (vitamins C, E and -carotene) might help protect against Alzheimer's disease, but this has not been supported in recent clinical studies. Another class of antioxidant chemicals, polyphenols, are now suspected. Polyphenols generally exist primarily in the skins of fruits and vegetables and are particularly abundant in teas, juices and wines.
The report appears in the September issue of The American Journal of Medicine. Full reference
http://www.eurekalert.org/pub_releases/2006-08/vumc-jmr082806.htm
http://www.eurekalert.org/pub_releases/2006-08/ehs-ssf082806.htm

High blood sugar linked to MCI and dementia

The first study to investigate the association over time between blood sugar and the risk of cognitive difficulties involved 1,983 post-menopausal women (mean age 67 years) and found that each 1% increase in their glycosylated hemoglobin level at the start of the four-year study period was associated with a 40% increased risk of developing MCI or dementia four years later. The glycosylated hemoglobin test gives a more stable measure of blood sugar level than the standard test, which measures blood sugar it the time of testing. A result of 7% or less indicates good long-term blood sugar control. Those with a level of 7% or more were four times more likely to develop MCI or dementia than women who tested at less than 7%.
The report appeared in the Journal of Nutrition, Health, and Aging. Full reference
http://www.eurekalert.org/pub_releases/2006-08/uoc--chb080906.htm

Brain enzyme treatment for Alzheimer's

In a new approach to treating Alzheimer’s, increasing brain levels of ubiquitin C-terminal hydrolase L1 (Uch-L1) — an enzyme that helps neurons rid themselves of excess or aberrant proteins — has restored a great deal of brain activity to mice with Alzheimer's symptoms. The enzyme Uch-L1 is part of a network that controls a memory molecule called CREB, which is inhibited by amyloid beta proteins in people with Alzheimer's. Uch-L1 is found at reduced levels in the Alzheimer's brain. As well as improving memory in genetically engineered mice, treatments that restored Uch-L1 levels corrected deficits in nerve transmission both in brain slices treated with amyloid-beta and in slices taken from transgenic mice.
The report appeared in the August 25 issue of Cell. Full reference
http://www.eurekalert.org/pub_releases/2006-08/cp-bet082106.htm
http://www.eurekalert.org/pub_releases/2006-08/cumc-crr082206.htm

Why chances of Alzheimer's increase with age

Experiments with roundworms have revealed two important proteins that help slow down the accumulation of amyloid-beta. HSF-1 breaks apart amyloid and disposes of it — but aging slows HSF-1, so it can't keep up. DAF-16 helps it out, by clumping extra amyloid together in a way that makes it less toxic. The finding supports recent research indicating amyloid clumps, or plaques, are not the main problem, rather, smaller amyloid tendrils inside cells are. The study also explains why aging increases the likelihood of Alzheimer’s. Most importantly of all, it suggests a new approach to treating Alzheimer’s.
The report was published online 10 August in Science Express, and in the 15 September issue of Science. Full reference
http://www.msnbc.msn.com/id/14290153/
http://www.boston.com/news/nation/articles/2006/08/11/scientists_find_natural_alzheimers_controls/

July

Predicting dementia risk

A large Scandinavian study has succeeded in calculating middle-aged people's chances of developing dementia later in life with 70% accuracy. The study confirms the importance of lifestyle factors. The study assessed factors such as blood pressure, body fat and cholesterol levels in 1,400 middle-aged Finns in the 1970s and 1980s, and found that those who at 40 were obese, or had high blood pressure, or high cholesterol levels, were twice as more likely to develop dementia by the age of 60. Having all three of these risk factors increased their chances six-fold. Other risk factors were age and low education.
The study was published online ahead of print on 3 August in Lancet Neurology. Full reference
http://society.guardian.co.uk/health/story/0,,1836464,00.html

Calorie restriction may help prevent Alzheimer's

A mouse study has found that beta-amyloid peptides can be reduced by restricting calorie intake, primarily through a low carbohydrate diet. Conversely, a high caloric intake based on saturated fat was shown to increase levels of beta-amyloid peptides. This is the first study to suggest that caloric restriction might inhibit the generation of beta-amyloid peptides, but there have been a number of studies providing evidence that high cholesterol, obesity, and other cardiovascular risk factors increase the likelihood of Alzheimer’s.
The study appeared in the August 4 issue of the Journal of Biological Chemistry. Full reference
http://www.sciencedaily.com/releases/2006/06/060614113128.htm

Apples fight memory loss

The study involved adult and old mice (some engineered to develop Alzheimer's-like symptoms) being fed either a standard diet, a nutrient-deficient diet, or a nutrient-deficient diet supplemented with apple juice concentrate. The mice on the apple juice-supplemented diet showed an increased production of acetylcholine in their brains and performed significantly better on maze tests. The amount of consumption was comparable to humans drinking approximately two 8 oz. glasses of apple juice or eating 2-3 apples a day. The findings also suggest that the apple-supplemented diet was most helpful in the framework of an overall healthy diet. Acetylcholine levels declined in both adult and old mice on the nutrient-deficient diet.
The study was published in the August issue of the Journal of Alzheimer's Disease. Full reference
http://www.sciencedaily.com/releases/2006/08/060801225922.htm

How mental and physical stimulation slows Alzheimer's

A new study reveals how mental and physical activity slows the cognitive decline seen in Alzheimer’s. In the study, genetically engineered mice were housed in either standard cages or ones with access to an enriched environment. After five months, the mice housed in the enriched environment had fewer Ab plaques, smaller plaque size, and reduced amyloid angiopathy compared to mice housed in standard cages. However there were no differences in the levels of soluble Ab peptide or the expression levels of its precursor protein (APP). Further investigation revealed differences suggesting that an enriched environment elicits protection via pathways that prevent Ab accumulation and enhance its clearance. The data confirm that an environment rich in mental and physical stimulation slows the progression of Alzheimer-like brain pathology.
The paper appeared in the August issue of the American Journal of Pathology. Full reference
http://www.sciencedaily.com/releases/2006/07/060724002331.htm

Weight Loss Precedes Dementia Diagnosis In Women

A study has come out finding that, in women, declining weight precedes dementia by many years. The retrospective study analyzed the medical records of 560 patients diagnosed with the onset of dementia between 1990 and 1994. The patients were matched with 560 controls. Among the women, average weight increased slightly over the preceding 30 years for the control group, but drifted downwards over the 30 years for those who developed dementia. The researchers suggest that changes in the brain interfered somehow with maintenance of body weight. The trend was not observed in men.
Findings were presented July 16 at the Alzheimer's Association International Conference on Alzheimer's Disease and Related Disorders in Madrid, Spain.
http://www.sciencedaily.com/releases/2006/07/060716090233.htm

Elders with dementia can tap into memory stores to give advice

Surprisingly, it appears that the best way to converse with an Alzheimer’s patient may be to ask them for advice. In two studies, researchers found that adults with moderate to severe symptoms of dementia can still be quite coherent and informative when asked for advice. In the first study, 14 people with early to advanced stages of dementia were asked about marriage, children and church in a purely social way, such as "Tell me about your children," and then later were asked for advice on the same topics, as in, "I'm thinking about having children. What kind of advice can you give me on that?" Patients were more coherent, informative and focused on the topic when asked for advice as opposed to when they were simply asked about their children, church or marriage. In the second study, six adults with dementia and six older adults without dementia, were given a booklet of pictures to guide them in teaching someone a simple recipe. Both groups successfully taught students to prepare the recipes, although those with dementia did need more prompting to finish the task.
Findings appeared in the June issue of The Gerontologist. Full reference
http://www.eurekalert.org/pub_releases/2006-07/fsu-sew071706.htm

Donezepil slows brain deterioration for some on road to Alzheimer's

According to a new study, the drug donepezil measurably (but still only slightly) slows the rate of hippocampal shrinkage in patients with mild cognitive impairment (a pre-Alzheimer's condition) who carried the apolipoprotein E4 (APOE 4) gene variant. The study involved 131 patients with mild cognitive impairment. For APOE 4 carriers, the rate of hippocampal atrophy was 4.5% per year, versus 6.14% in placebo-treated patients. Rates of shrinkage for cognitively normal people in their late 70s are approximately 1.4 percent per year. Vitamin E had no significant effect on atrophy for any patients.
Findings were presented July 17 at the Alzheimer's Association International Conference on Alzheimer's Disease and Related Disorders in Madrid, Spain.
http://www.eurekalert.org/pub_releases/2006-07/mc-msd071306.htm

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